2 edition of Membrane abnormalities in hypertension found in the catalog.
Membrane abnormalities in hypertension
Includes bibliographies and indexes.
|Statement||editor, Chiu-Yin Kwan.|
|Contributions||Kwan, Chiu-Yin, 1947-|
|LC Classifications||RC685.H8 M38 1989|
|The Physical Object|
|Pagination||2 v. :|
|ISBN 10||0849345278, 0849345286|
|LC Control Number||88026305|
Anti-glomerular basement membrane diseases (anti-GBM diseases) is a rare disorder that can involve quickly worsening kidney failure and lung disease. Some forms of the disease involve just the lung or the kidney. Anti-GBM disease used to be known as Goodpasture syndrome.
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First Published inthis two-volume set offers a full insight into membrane abnormalities during a state of hypertension. Carefully compiled and filled with diagrams, references and information this set is recommended for students of medicine and other professionals in their respective fields.
Membrane abnormalities in hypertension. [Chiu-Yin Kwan;] Home. WorldCat Home About WorldCat Help. Search. Search for Library Items Search for Lists Search for Book: All Authors / Contributors: Chiu-Yin Kwan. Find more information about: ISBN:. The aim of this review is to outline some interrelationships between abnormal lipid metabolism and altered membrane structure or function in human and experimental hypertension, and to evaluate whether abnormal lipid metabolism, which is responsible for a great part of membrane abnormalities found in hypertension, might be an integral part of Cited by: Journals & Books; Help Download PDF and pharmacologic interventions aiming to normalize abnormal lipid metabolism could be useful for amelioration of membrane abnormalities and lowering of high blood pressure.
Future studies of functional membrane alterations in hypertension or dyslipidemia will therefore require the detailed determination Cited by: Membrane Abnormalities in Essential Hypertension. One of them is this Membrane Abnormalaties In Hypertension.
Kelly Mays: The guide with title Membrane Abnormalaties In Hypertension contains a lot of information that you can find out it.
You can get a lot of help after read this book. This book exist new know-how the information that exist in this reserve represented the condition of the world. During the past 15 years extensive research in experimental and clinical hypertension has indicated that there are cell membrane abnormalities associated with chronically elevated arterial pressure.
Many aspects appear to be altered: cation concentration, cation permeability, active, co- and countertransport, and calcium binding are some. Membrane Skeleton Genetic Hypertension Renal Brush Border Membrane Vesicle Primary Genetic Abnormality Primary Arterial Hypertension These keywords were added by machine and not by the authors.
This process is experimental and the keywords may be. Hypertension (High Blood Pressure): Selected full-text books and articles Fast Facts about Hypertension Nutrition Health Review, No. 96, Winter Read preview Overview. Drawing on findings from many laboratories, this chapter will review evidence for the induction of abnormalities of vascular K+ and Ca2+ channels during hypertension and discuss mechanisms that.
adshelp[at] The ADS is operated by the Smithsonian Astrophysical Observatory under NASA Cooperative Agreement NNX16AC86A. Purchase Comprehensive Hypertension - 1st Edition. Print Book & E-Book.
ISBNPregnancy complicated by hypertension is commonly associated with placental insufficiency, thereby resulting in fetal growth retardation.
Furthermore, reduced utero-placental blood flow has been recognized in cases of severe preeclampsia with hypertension. Thus, it. SUMMARY To test the hypothesis that structural abnormalities exist in the cell membrane in persons with essential hypertension and that these abnormalities affect membrane-related cellular functions, we examined several membrane-dependent phenomena and membrane lipid composition in the blood cells of subjects with essential hypertension.
In addition, it was shown that the reduced membrane fluidity of RBCs was correlated with higher plasma hs-CRP and lower plasma NO metabolite levels, indicating that abnormalities in RBC membranes might be associated with increased inflammatory status and endothelial dysfunction in hypertension.
Cell membrane abnormalities and genetic hypertension. Bianchi G, Ferrari P, Cusi D, Guidi E, Salardi S, Torielli L, Tripodi MG, Niutta E, Elli A, Vezzoli G, et al.
Evidence indicates that an alteration in kidney function has a causal role in the pathogenesis of hypertension in.
Evidence presented in the current review suggests that hypertension is associated with a generalized membrane defect. Abnormalities in ion transport in vascular smooth muscle cells are the most relevant to the pathogenesis of hypertension since they could.
a LANGE medical book CURRENT ESSENTIALS: NEPHROLOGY & HYPERTENSION Edited by Edgar V. Lerma, MD Clinical Associate Professor of Medicine Section of Nephrology. To test the hypothesis that structural abnormalities exist in the cell membrane in persons with essential hypertension and that these abnormalities affect membrane-related cellular functions, we examined several membrane-dependent phenomena and membrane lipid composition in the blood cells of subjects with essential hypertension.
Amnion bands and loss of amnion membrane: infant abnormalities such as loss of finger should always have high intensity search for bands in placenta. Fetal & maternal RBCs & WBCs: there should be very few NRBCs in villous circulation of a normal term birth; if found, consider causes of hypoxia, alpha Thal., hemolytic problems.
Hypertension, also called high blood pressure, condition that arises when the blood pressure is abnormally high. Hypertension occurs when the body’s smaller blood vessels (the arterioles) narrow, causing the blood to exert excessive pressure against the vessel walls and forcing the heart to work harder to maintain the pressure.
Although the heart and blood vessels can tolerate increased. These results suggest a genetic and functional link in MHS between cell membrane cation transport abnormalities and hypertension.
Thus, erythrocyte cell membrane may be used for approaching the problem of defining the genetically determined molecular mechanism underlying the development of a type of essential hypertension.
35 references.}, doi. Strip of membrane, including the point of membrane tear, should be obtained Beginning with the point of tear, the strip is rolled around a long, thin probe with the amnion facing inward Roll is then fixed for a minute with an acidic fixative (e.g.
acetic acid) and sectioned. Search the world's most comprehensive index of full-text books. My library. Membrane Receptors, Channels and Transporters in Pulmonary Circulation is a proceeding of the Grover Conference (Lost Valley Ranch and Conference Center, Sedalia, Colorado; September), which provided a forum for experts in the fields of those receptors, channels and transporters that have been identified as playing key roles in the physiology and pathophysiology of the pulmonary.
Clinical Disorders of Membrane Transport Processes is a softcover book containing a portion of Physiology of Membrane Disorders (Second Edition).
The parent volume contains six major sections that deal with general aspects of the physiology of transport processes and specific aspects of transport processes in cells and in organized cellular.
Hemolytic anemias due to abnormalities of the erythrocyte membrane comprise an important group of inherited disorders. 1, 2 These include hereditary spherocytosis (HS), hereditary elliptocytosis (HE), hereditary pyropoikilocytosis (HPP), and the hereditary stomatocytosis (HSt) syndromes.
These disorders are characterized by clinical and laboratory heterogeneity and, as. About the Book; Book Chapters. Section I: Diseases and Disorders. Abdominal Compartment Syndrome; Pulmonary Hypertension (Arterial) Pulmonary Lymphoid Granulomatosis; Pulmonary Nodules; Pulmonary Thromboembolism; (Nictitating Membrane) Abnormalities; Thromboembolism; Thyroid Hormone Alterations; Tonsillar Disorders.
Middle ear disorders may be secondary to infection, eustachian tube obstruction, or trauma. Information about objects placed in the ear and symptoms such as rhinorrhea, nasal obstruction, sore throat, upper respiratory infection, allergies, headache, systemic symptoms, and fever aid in making a appearance of the external auditory canal and tympanic membrane often yields a diagnosis.
A study of Ca-ATPase and Ca-pump activity of the erythrocyte membrane's inverted vesicles with different cytoskeletal protein status demonstrated that Ca-pump activity remained unchanged, and Ca-ATPase activity was increased by %, as compared to. Abnormalities of any of the mechanisms involved in the maintenance of normal BP, including sodium intake, the renin-angiotensin-aldosterone mechanism, and sympathetic nervous system (SNS) stimulation, can result in the development of hypertension.
primary hypertension. Abnormalities of glucose, insulin, and lipoprotein metabolism are common in. Since there have been major advances in our understanding of the genetic and genomics of pulmonary arterial hypertension (PAH), although there remains much to discover. Based on existing knowledge, around 25–30% of patients diagnosed with idiopathic PAH have an underlying Mendelian genetic cause for their condition and should be classified as heritable PAH (HPAH).
is a rapid access, point-of-care medical reference for primary care and emergency clinicians. Started inthis collection now contains interlinked topic pages divided into a tree of 31 specialty books and chapters.
Purchase Membrane Transporters in the Pathogenesis of Cardiovascular and Lung Disorders, Volume 83 - 1st Edition. Print Book & E-Book. ISBN Buy Disorders of Blood Pressure Regulation: Phenotypes, Mechanisms, Therapeutic Options (Updates in Hypertension and Cardiovascular Protection): Read Books Reviews - Pulmonary hypertension is a common complication in lung disease.
In the most recent revised classification of pulmonary hypertension (PH), chronic lung diseases or conditions with alveolar hypoxia are included in WHO Group III of PH-related diseases (Table 1) [1,2].In this classification the structure of this group was for the most part unchanged.
Placental AbnormalitiesPlacental Abnormalities -- Abnormal Shape or Implantation-Abnormal Shape or Implantation- Abnormality Definition Clinical significance Membranaceous Placenta all of the fetal membranes are covered by functioning villi and the placental develops as a thin membranous structure occupying the entire periphery.
Primary hypertension has multiple or unknown causes, and secondary hypertension has known causes. In most cases of primary hypertension, increased peripheral resistance is the reason for the elevated pressure.
Even moderate elevation of arterial blood pressure is associated with a shortened life expectancy. Laminar necrosis was observed in 27% of placentas from mothers with hypertensive disorders (preeclampsia or chronic hypertension) and was linked to other maternal, fetal, neonatal, and placental conditions known to be associated with perinatal hypoxia, and with hypoxia-inducible factor, apoptosis, and oxidative stress playing role in the.
Pulmonary hypertension (PH) is a severe haemodynamic disorder in which the pulmonary artery pressure is persistently elevated, leading to right-sided heart failure and death. Recently, chronic myeloproliferative diseases associated with pulmonary hypertension were included in the group 5 category, corresponding to PH for which the aetiology is unclear and/or multifactorial.
Hypertension is a worldwide problem that affects up to 22% of adults and contributes to the global burden of disability due to cardiovascular disease. Several factors influence blood pressure and participate to the development of hypertension.
Among these factors, polyunsaturated fatty acids of the omega-3 family (omega-3 PUFA) are effective hypotensive agents.Heritable pulmonary arterial hypertension.
The study of genetics in pulmonary hypertension began in when D resdale et al.  first described familial pulmonary arterial hypertension (PAH), suggesting the potential role of genetic defects in the development of the the availability of modern genetic tools, genealogic studies allowed us to understand the genetic transmission.Diabetic nephropathy is the most common cause of nephrotic syndrome in adults.
Diabetic nephropathy is also the most common cause of end-stage renal disease in the US, accounting for up to 80% of cases. The prevalence of renal failure is probably about 40% among patients with type 1 diabetes prevalence of renal failure among patients with type 2 diabetes mellitus is usually stated.